Dissimilar to the others, it occurs in the case that the lesion is present on the border between medial and posterior, or anterior and medial. If the lesion is in front, the patient is able to understand what is said but never speaks. If it\u2019s at the back, it would be like Wernicke\u2019s aphasia. If it\u2019s located on both sides, it would be a transcortical mixed aphasia. Regardless of the type it is of, this is an aphasia in which repetition is perfectly preserved. Its communication with other networks is impaired, although it is only the language networks that is not impaired. That\u2019s why repetition is preserved.<\/p>\n
The patients lose the ability of initiative. They have impaired speech, but they don\u2019t care. They have a tongue-tied speech, while having relatively preserved comprehension and repetition. Even the patients who do not speak at all can easily repeat a word said to them.<\/p>\n
Despite having troubles in finding words, the patients speak fluently. Although the don\u2019t understand what they are told, they can fluently repeat what was said.<\/p>\n
It is similar to global aphasia. There is a damage separating the language area from the other parts of the brain. The language area remains robust in itself. Thus, the patient is able to repeat and analyze sounds. These patients have a tongue-tied speech, impaired comprehension, though repetition is preserved.<\/p>\n
Anomia means \u201cbeing unable to name\u201d. It could be said that it is the best type of aphasia. These patients can talk fluently and understandably. In contradiction to Wernicke\u2019s aphasia, they have a decent comprehension and don\u2019t have verbal or neo-logistic paraphasia. Nonetheless, the speeches of these patients lack objective nouns. For instance, in order to say \u201cCould you pass me the glass inside the window?\u201d they say \u201cCould you pass me the thing inside the thing?\u201d When a naming examination is carried out thought the demonstration of the objects one by one, it can be seen that this skill is extremely impaired. The patients cannot say the name of the object, instead, they describe the object. Like \u201cwe write with this thing\u201d, \u201cit is used to drink water\u201d. However, the patient has a normal repetition. These patients cannot find the word, they don\u2019t remember the name of the objects. Having quite an impaired naming, they generally say \u201cthing\u201d. Besides, their comprehension, reading and writing are preserved.<\/p>\n
Anomic Aphasia is not a common syndrome with this typical form it has. On the other hand, all Wernicke\u2019s aphasias pass through the anomic aphasia level in one phase of the recovery. Although anomic aphasia is generally caused by temporo-occipital lesions, or temporo-parietal lesions extending to the angular gyrus (where reading and writing are also heavily impaired), anomic aphasia can also be seen in different anatomic localizations (in this case, the patient\u2019s reading and writing may remain intact).<\/p>\n","protected":false},"excerpt":{"rendered":"
Dissimilar to the others, it occurs in the case that the lesion is present on the border between medial and posterior, or anterior and medial. If the lesion is in front, the patient is able to understand what is said but never speaks.<\/p>\n","protected":false},"author":2,"featured_media":0,"comment_status":"open","ping_status":"closed","sticky":false,"template":"","format":"standard","meta":{"_acf_changed":false,"footnotes":""},"categories":[501],"tags":[],"class_list":["post-1263","post","type-post","status-publish","format-standard","hentry","category-articles"],"acf":[],"yoast_head":"\n